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| Bibliography of Selected Scientific Research on the Health Effects of MercuryAccumulation from Dental Amalgam Bibliography of Selected Scientific Research on the Health Effects of Mercury Accumulation from Dental Amalgam A THE ABSORPTION OF MERCURY FROM DENTAL FILLINGSFritz L. Lorscheider, Ph.D. and Murray S. Vimy, DDS. University ofCalgary Medical School, Alberta, Canada.Drs. Lorscheider and Vimy have shown definitively that mercury iscontinuously released from amalgam fillings, both as vapor and in microscopic particles,once the fillings are placed in the teeth. The mercury emitted from the fillings istransported to every part of the body via the air pathways, the digestive tract and theMood stream, and accumulates in tissues and organ systems.1. Dental Amalgam Mercury: Background. (A summary of research results on dental amalgam mercury to date.)M. J. Vimy andF. L. Lorscheider, Faculty of Medicine and Medical Physiuology, University of Calgary,Calgary, Alberta. May, 1993.2. Dental"Silver" tooth fillings: a source of mercury exposure revealed by whole-bodyimage scan and tissue analysis. ByLeszek J. Hahn, Reinhard Woiber, Murray J. Vimy, Yoshimi Takahashi, and Fritz L.Lorscheider. FASEB Journal, Vol.3, Dec. 1989. pp.2641-2646.3. Whole-body imaging of the distributionofmercury released from dental fillings into monkey tissues.By Leszek J. Hahn, ReinhardKloiber, Ronald W. Leininger, Murray J. Vimy, and Fritz L. Lorscheider.FASEB Journal, Vo.4, Nov.1990, pp.3256-3260.4. Mercury from dental "silver" tooth fillings impairs sheepkidney function. By N.D. Boyd,H.Benediktsson, M.J. Vimy, D.E. Hooper, and F. L. Lorscheider. American Journal ofPhysiology, No.261, 1991, pp. R1O1O-1014.5. Maternal-fetal distribution of mercury (203HG) released from dentalamalgam fillings. By M.J. Vimy, Y.Takahashi, and F.L. Lorscheider. American Journal of Physiology, No.258, 1990, pp.R939-945.6. SYMPOSIUM OVERVIEW: Toxicity Assessment of Mercury Vapor from DentalAmalgams. By Peter L. Goering, W. DonGalloway, Thomas W. Clarkson, Fritz L. Lorscheider, Maths Berlin and Andrew S. Rowland.Journal of Fundamental and Applied Toxicology, vol.19, 1992, pp.319-329.7. Evaluation ofthe safety issue of mercuryrelease from dental fillings, Fritz L.Lorscheider and Murray J. Vimy. The FASEB Journal, Vol.7, December 1993, p1432-1433.8. ADP-Ribosylation of Brain Neuronal Proteins Is Altered by In Vitroand In Vivo Exposure to Inorganic Mercury. Pawel Palkiewicz, Henk Zwiers, and Fritz L. Lorscheider. Journal ofNeurochemistry,, Vol.62, No.5, 1994, pp.2049-2052 B. BASIC PHYSIOLOGICAL RESEARCH ON THE RELATIONSHIP OF BRAIN MERCURY ACCUMULATIONS FROM DENTAL AMALGAM MERCURY TO ALZHEIMER'S DISEASEWilliam R. Markesbery, M.D., William D. Ehmann, M.D., (andcolleagues at the University of Kentucky's Sanders-Brown Center on Aging).Drs. Markesbery's and Ehmann's experiments have shown that there arehigher concentrations of mercury in the autopsied brains of patients who died ofAlzheimer's than are present in the autopsied brains of patients who did not haveAlzheimer's. In the Alzheimer's patients' brains, there are also lower concentrations ofselenium and zinc, the two chief mineral antagonists of mercury. Markesbery and Ehmannhave also demonstrated that there are higher concentrations of mercury in the brains ofpeople who have more and larger amalgam dental fillings. Additional research is underwayto link the presence of amalgam mlings more closely with the incidence of Alzheimer's. 9. Trace element imbalances in isolated subcellular fractions ofAlzheimer's disease brains. By DavidWenstrup, William D. Ehmann, and William R. Markesbery. Brain Research, No 533, 1990, pp.125-130.10. Mercury imbalances in patients with neurodegenerative diseases. W. D. Ehmann, E. J. Kasarskis, and W. R.Markesbery. (In Press) C. MERCURY AS A CAUSE OF TUBULIN TANGLES SIMILAR TO THOSE FOUND IN ALZHEIMER'S DISEASE, AND THE DEVELOPMENT OF A NON-INVASIVE TEST FOR ALZHEIMER'S Boyd E. Haley, Ph.D. (and colleagues.) Professor of Medical Chemistryand Biochemistry, Markey Cancer Center, University of Kentucky.Dr. Haley has produced tubulin defects in laboratory cultures of braintissue by adding a low concentration of mercury plus EDTA, a common food additive. Tubulindefects are thought to be the mechanism which produces the neuro fibrillary tanglescharacteristic of Alzheimer's Disease. Haley has also identified the first biochemicalmarker for Alzheimer's, that is, an enzyme found in Alzheimer's patients cerebro-spinalfluid which is not found in normal persons and which could be used as one basis ofdiagnosis for the disease. 11. HG2+ induces GTF-Tubulin interactions in rat brain similar to thoseobserved in Alzheimer's Disease. E. Duhr, C. Pendergrass, E. Kasarskis, J. Slevin & B.Haley. FASEB Journal, 1992. Abstract Dated December 3, 1991.12. HgEDTA Complex InhibitsGTP Interactions With The E-Site of Brain Beta-Tubulin. Edward F. Duhr, James C. Pendergmass, John T. Slevin, and Boyd E. Haley. Toxicol.Appl. Pharmacol., 1993.13. DMSA acid partially restores tubulin intereactions to bothAlzheimer's Diseased brains and to HGEDTA treated control brains.J.C. Pendergrass, E.F. Duhr, J.T. Slevin, and B. E. Haley.Experimental Biology 93 Abstract, dated November 17, 1992.14. Aberrant Guanosine Triphosphate-Beta- Tubulin Interaction inAlzheimer's Disease. Sabiha Khatoon,Ph.D., Susan R. Campbell, B.S., Boyd E. Haley, Ph.D.and John T. Slevin, M.D. Annals ofNeurology, Vol.26, No.2, August 1989, pp. 210-215.15. Detection of glutamine synthetase in the cerebrospinal fluidofAlzheimer diseased patients: A Potential diagnostic biochemical marker. Debra Gunnersen and Boyd Haley. Proceedings of theNational Academy of Science, Vol.89, pp. 11949-11953, December 1992, Biochemistry. D. MERCURY CHELATING AGENTS AND MERCURY ABSORPTION MEASUREMENT H. Vasken Aposhian, Ph.D., University Department of Molecular andCellular Biology, University of Arizona, Tucson, Arizona.Dr. Aposhian has established, through carefully constructedexperiments, that in people who have amalgam fillings two-thirds of the total body burdenof mercury is the product of mercury absorption from amalgam dental fillings. He has alsocreated a scoring system based upon the number of fillings and the number of surfaces onthe fillings, quantifying the expression of the amount of dental amalgam in anindividual's mouth. Finally, he has also been experimenting with chelating agents whichmight remove mercury accumulations from the body.16. Urinary mercury after administration of 2,3 dimercaptopropane- 1-sulfonic acid: correlation with den -tal amalgam score. H. Vasken Aposhian, David C. Bruce, Wilfred Alter, Richard C. Dart, Katherine M.Hurlbut, and Mary M. Aposhian. The FASEB Journal, Vo. 6, April 1992, pp. 2472-2476.17. DMSA and DMPS-Water soluble antidotes for heavy metal poisoning. H. Vasken Aposhian. American Review of Pharmacologyand Toxicology, 1983, Vol.23,193-215.18. Determination andMetabolism ofDithiol Chelating Agents, XII. Metabolism and Pharmacokinetics of Sodium 2,3-Dimercap top ropane-2-Sulfonate in Humans. Richard M. Maiorino, Richard C. Cart, Dean E. Carter and H. Vasken Aposhian. TheJourmal of Pharmacology and Experimental Therapeutics, 1991, Vol. 259 No.2, pp.808-814. 19. MESO-2, 3 -DIMERCAPTOS UCCINIC ACID:Chemical, Pharmacological and Toxicological Properties of an Orally Effective Metal Chelating Agent. H. Vasken Aposhian and Mary M. Aposhian. E. MERCURY AND RENAL DISEASEJames S. Woods, Battelle Seattle Research Center and the University ofWashington, Seattle, Washington, and colleagues.Dr. Woods has been investigating how kidney tissue is damaged by thepresence of maercury. He has also identified ways in which porphyrin relationships aredisrupted by the presence of mercury, which could provide another type of marker to testfor the presence of mercury in body tissues.20. Mercury-induced H202 production and lipid per-oxidation in vitro inrat kidney mitochondria. Bert-Ove Lind,Dennis M. Miller and James S. Woods. Biochemical Pharmacology, Vol.42, 1991, Suppl. pp.S181-S187. Pergamon Press.21. Urinary Porphyrin Profiles as Biomarkers of Trace Metal Exposureand Toxicity: Studies on Urinary Prophyrin Excretion Patterns in Rats during ProlongedExposure to Methyl Mercury. James S. Woods, Miriam A. Bowers, and Holly A. Davis.Toxicology and Applied Pharmacology, Vol.110, 1991, pp.464-476.22. Enhancement of Gamma-Glutamylcysteine Syn-thetase mRNA in RatKidney by Methyl Mercury. James S.Woods, Holly A. Davis, and Robert P. Baer. Archives of Biochemistry and Biophysics, Vol.296, No.1, July 1992, pp. 350-353.23. QuantitativeDetermination of Porphyrins in Rat and Human Un tie and Evalution of Urinary ProphyrinProfiles during Mercury and Lead Exposures. Miriam A. Bowers, Lauri J. Aicher, Holly A. Davis, and James S. Woods. TheJournal of Laboratory and Clinical Medicine, St. Louis. Vol.120, No.2, pp.272-281, August,1992.F. ANTIBIOTIC RESISTANCEAnne 0. Summers, Ph.D., Department of Molecular Biology andMicrobiology, University of Georgia, Athens, GeorgiaDr. Summers has been engaged in research on gastrointestinal tractbacteria and their resistance to antibiotics, which is a serious and widespread medicalproblem. With the collaboration of Drs. Lorscheider and Vimy and Dr. Stuart Levy at TuftsUniversity, she has demonstrated that bacterial resistance to antibiotics can be createdby exposing g.i. tract bacteria to mercury, such as that absorbed into the g.i. tractthrough the presence of amalgam dental fillings in the mouth.24. "Silver"Dental Fillings Provoke An Increase in Mercury and Antibiotic Resistant Bacteria in theMouth and Intestines of Primates. Anne0. Summers, Murray Vimy and Fritz Lorscheider, The Alliance for the Prudent Use ofAntibiotics (APUA) Newsletter, Fall 1991, Vol.9, No.3, pp.4-5.25. Mercury Released from Dental "Silver" Fillings provokesan increase in Mercury- and Antibiotic-Resis-taut Bacteria in Oral and Intestinal Flora ofPrimates.Anne 0. Summers, Joy Wireman, Murray J. Vimy, Fritz L.Lorscheider, Bonnie Marshall,Stuart B. Levy, Sam Bennett, and Lynne Billard. Antimicrobial Agents and Chemotherapy, April 1993, Vol.37, No.4, p.825-834. G. CLINICAL MEDICAL TREATMENT OF DENTAL-FILLING MERCURY TOXICITYAlfred V. Zamm, M.D., FACA, FACP, ill Maiden Lane, Kingston, New York 12401-4597 Reports on clinical treatment of a wide variety of illnesses linked to dentalamalgam mercury toxicity by a dermatologist and allergy specialist.26. Candida Albicans Therapy: is there ever an end to it? Dental Mercury Removal: aneffective adjunct. Alfred V. Zamm,M.D., FACA, FACP. Journal of 0rthomolecu-lar Medicine, Vol.1 No.4, pp.261-266.27. Dental Mercury: A Factor That Aggravates and Induces Xenobiotic Intolerance. Alfred V. Zamm, M.D., FACA, FACP. Journal ofOrthomolecular Medicine, Vo. 6 No.2, Second Quarter 1991, pp.67-77.28. Mercury and Dentistry: What the Sensitive Patient Should Know.Alfred V. Zamm, M.D., FACA, FACP. The Mercury in Medicine andDentistry Newsletter Quarterly, Vol.1 No.1, Autumn 1986, pp.1-8.29. Removal of Dental Mercury: Often an Effective Treatment for the Very SensitivePatient. Alfred V Zamm, MD, FACA, FACP. Journal ofOrthomolecular Medicine, Vol.5, No.3, Third Quarter 1990, pp.138- H. RESEARCH ON THE IMPACT OF DENTAL MERCURY ON THE IMMUNE SYSTEM, BRAIN, AND NEUROLOGICAL SYSTEM. David Eggleston, D.D.S.30. Effect of dental amalgam and nickel alloys on T-lymphocytes: preliminary report. Eggleston, David W. DDS. Journal of prostheticdentistry: May 1984, Vol 31, No.5.31. Correlation of dental amalgam with mercury in brain tissue.Eggleston David W. DDS, Magnus Nylander DDS, et al. J. Pros.Dent. 58:704-7,198732. Dental Amalgam: A Review of the Literature. Eggleston, DW: Compend. Cont. Ed. Dent. Vol. X, No.9. |
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